Complications of Myocardial Infarction

Complications of Myocardial Infarction

The complications of a myocardial infarction are as follows :
  • Arrhythmias – These usually occur within the first 48 hours following an infarct and may be life-threatening. They are due to myocardial irritability caused by lack of oxygen (ischaemia), release of potassium and calcium from dead cells and disturbances to the conductive mechanism of the heart. This is particularly true when the area around the sinoatrial or atrioventricular nodes is affected. Myocardial irritability is identified by the number of ventricular ectopics seen. There is a danger that an ectopic may fall on the T wave (r on T) and ventricular fibrillation ensues.
  • Heart failure – Backpressure from the ventricle affected onto the atrium on the same side exists due to the ventricle being unable to expel the blood in it effectively. This will cause backpressure on either the pulmonary circulation (causing pulmonary oedema) or the systemic circulation (peripheral in organ oedema). The cautious use of diuretics and vasodilators may minimise this.
  • Pericarditis – This is thought to be caused by an autoimmune response initiated by the necrotic tissue within the heart causing an inflammatory reaction in the pericardium. The symptoms of this are similar to the infarct though the pain is longer lasting and a pericardial rub may be heart on auscultation.
  • Mitral regurgitation – This is due to either papillary muscle infarct or rupture that causes the mitral (left side) valve to become incompetent. This will worsen any cardiac failure by allowing blood to enter the left atrium during systole.
  • Myocardial rupture – This is due to the weakened area of necrotic myocardium and is likely to occur within seven days post infarct. It is more often seen in patients with a transmural infarct (infarct extends through all the layers of the heart) and may cause a cardiac tamponade. An intraventricular septal rupture can occur within 2 weeks following the infarct and is a very serious complication. It allows the flow of blood from the left to right sides of the heart and will flood the lungs with pulmonary oedema. The blood entering the aorta will also be severely diminished causing cardiogenic shock. The combination of these is likely to kill the patient outright depending on the size of the rupture. Emergency surgery is the only treatment for this.
  • Myocardial aneurysm – This is the ballooning out of the infracted myocardial tissue into the chamber affected. This may seriously reduce the cardiac output and will cause the heart to contract in an abnormal fashion. The size of this can be identified my echocardiography. Surgery is the only treatment.
  • Emboli – These form when clots formed in the necrotic areas break free and enter the bloodstream. Pulmonary, cerebral or deep vein thrombosis may form. Also further myocardial infarction may occur if the emboli enter the coronary arteries. Emboli may or may not kill the patient outright, it will depend on the location and size of the vessel they are blocking. Anticoagulant drugs such as heparin and aspirin will be given and the patient closely monitored for further emboli or moving of the emboli to other areas.
  • When the patient is discharged from the coronary care / high dependency unit to a medical ward, their lifestyle is examined and teaching about how to change their lifestyle to one that will minimise the risk of a repeat infarct will begin. Involvement with a dietician is essential in order for the patient to start to look at their diet and alcohol intake.


Reference :
http://www.nursingtheory.nhs.uk

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